Teething in children: modern concepts


Normally, a person grows 52 teeth over a lifetime: 20 milk teeth and 32 permanent teeth. Supernumerary teeth are “extra” dental units that appear in addition to some incisors, canines, premolars or molars. In various medical sources, the phenomenon is called hyperdontia, supradontia or polyodontia. A superset can include 1–2 teeth or reach several hundred dystopic structures, be true or false.

Supernumerary teeth: causes of anomaly

The main cause of polyodontia is a violation of the formation or development of tooth germs. Doctors are considering several theories for this phenomenon:

  1. Heredity – hyperdontia develops as a result of genetic abnormalities that can be transmitted from parents to children. In this case, the pathology should manifest itself in the patient’s closest blood relatives.
  2. Splitting of the root germ of the tooth in the embryonic period of development. Usually observed after the mother becomes ill during pregnancy. Characteristic of single supernumerary teeth (1–2 pcs.).
  3. Failure during the embryonic period of tooth formation with the formation of multiple dental buds (up to several hundred). It is observed when the mother lives in unfavorable environmental conditions, during treatment with dangerous drugs, taking drugs, or alcohol abuse.

In more rare cases, polyodontia develops as a result of odontoma, a benign tumor in the area of ​​the jaw arches. The disease is typical for children and young people during the period of active growth of tooth germs. Such a tumor structure may contain several tens or even hundreds of highly deformed dentin-enamel formations.

Provoking factors in the prenatal period:

  • malfunction of genes - primarily the Msx1 gene (responsible for the formation of tooth germs);
  • viral infections;
  • taking teratogenic and potentially dangerous drugs;
  • smoking, drug use, alcohol use;
  • poor environmental conditions;
  • exposure to radiation - not only direct exposure, but also increased general background radiation in the place of residence/work.

Reference! Some scientists consider the presence of supernumerary teeth as an atavism. Thus, it has been scientifically proven that various species of the genus Homo could boast a set of teeth of 36–44 pieces. However, this theory does not explain in any way the appearance of multiple rudiments covering all spaces of the sky (a typical example is an Indian boy who had to remove 230 supernumerary units).

Polyodontia is an abnormal number of teeth.

In medicine, this disease is often called hyperdontia, and “extra” dental elements are called supernumerary teeth. Research is still being conducted into why this pathology occurs. Most scientists associate it with disturbances in the formation of tooth germs.

Nature provides that a person grows no more than 20 milk teeth and 32 permanent teeth in a lifetime, but exceptions occur, and in our time quite often. According to statistics, on average, dental anomalies occur in 2% of the world's population, most often in men.

In 2014 alone, two operations were performed, in one of which 80 teeth were removed, and in the other, a record 232 teeth. Until this time, the maximum figure was 37 teeth.

The most common hyperdontia (anomaly in the number of teeth) is an anomaly of the upper incisors. Supernumerary teeth are less common among the lower incisors and in other parts of the jaw. They can come in a wide variety of shapes and sizes. These are usually small, cone-shaped teeth.

Extra teeth lead to deformation of the dentition, so it is recommended to remove supernumerary elements. Another reason for removal is that most patients with this pathology have a lisp.

The formation of extra teeth is quite common today. According to statistics, 70% of patients have only one extra incisor, in 25% of cases – 2 supernumerary elements, and only 5% of all patients have 3 or more teeth during examination.

Symptoms of pathology


The main symptom is the presence of teeth beyond the natural set. They can have different positions, shapes and numbers. The process of teething of abnormal teeth is often accompanied by pain, fever, and inflammation of areas of the oral cavity. If measures are not taken in a timely manner, complications may develop:

  • redness and swelling at the site of the impacted tooth;
  • problems with chewing food and digestion;
  • injuries to the mucous tissues of the oral cavity;
  • violation of the position of the main teeth (dystopia) and the formation of malocclusion;
  • various speech defects - mainly problems with the pronunciation of hissing sounds;
  • loosening of adjacent normal teeth;
  • deformation of the jaw bones.

In young children and adults, the pathology has its own nuances. They are associated with age-related characteristics of the body and affect the subsequent development of maxillofacial structures. Statistics on the spread of pathology in children and adults:

  • 60% of cases are children and adolescents during the period of replacement of milk teeth with permanent ones;
  • 35% of cases are adults;
  • 5% of cases are small children during the period of growth of baby teeth.

Reference! Supernumerary teeth, as a rule, have a standard structure, sometimes with a smaller crown size. Less common are deformed formations of a teardrop-shaped, lumpy, chisel-shaped form.

Prevention of dystopia

Dentists are confident that if you carefully monitor your child’s dental health from early childhood, you can avoid most bite problems. Dystopic teeth are no exception, because their appearance is not always associated with hereditary factors. To prevent dystopia, doctors recommend the following:

  • proper nutrition, adherence to the daily routine of a pregnant mother;
  • prevention of jaw injuries from an early age;
  • regular visits to the dentist, wearing devices to correct malocclusion;
  • giving up bad habits (including weaning off the pacifier after a year and from thumb sucking in sleep), etc.

If you and your child regularly visit a dentist, he will give recommendations on what exactly to do in your case.

Features of polyodontia in children

Possible symptoms of hyperdontia in childhood:

  • intrauterine formation of teeth (a child is born with several teeth);
  • the appearance of teeth in the first months, weeks and even days of life;
  • delayed eruption of baby teeth.

This has unpleasant consequences:

  • problems with feeding the baby - he does not latch onto the nipple well and sucks weakly;
  • swelling of the mucous tissues can spread to the nasal area and cause breathing problems;
  • poor closure of the dental arches causes severe salivation;
  • a prolonged increase in temperature to 38C is possible.

Hyperdontia is especially harmful during the period of speech formation. Teeth located outside the general row prevent the tongue from taking the correct position when pronouncing most consonant sounds. Without eliminating the pathology, any speech therapist will be powerless.

In addition to true hyperdontia, children (less often adults) can develop the so-called false form of the disease. It is characteristic of the period of tooth change and develops during the eruption of molars next to the milk teeth that have not yet fallen out. This is a fairly common phenomenon that usually goes away on its own as the dentition finally forms. In some cases, orthodontic treatment may be required.

Dystopic wisdom tooth: what is it?

A dystopic wisdom tooth is a third molar, also known as a “figure eight”, which is positioned incorrectly relative to the rest of the dentition. Such a tooth almost always needs to be removed.

A special case of a dystopic wisdom tooth is an unerupted, but fully formed figure eight, which turns out to be turned parallel to the gum. In this case, the tooth is there, although it is not visible, and it is in an incorrect position, so we can talk about dystopia.

Two examples of dental dystopia

Features of polyodontia in adults and adolescents

In adults, after complete replacement of complete teeth, the presence of additional units can lead to additional pathologies:

  • chronic rhinitis, sinusitis when the wall of the maxillary sinuses is perforated by the roots of supernumerary impacted structures;
  • interdental caries - due to teeth fitting too closely to each other.

Retention and dystopia


Adults are characterized by 2 main types of supernumerary teeth:

  • Dystopic teeth are the name given to teeth with deviations in the direction of growth. The peculiarities of the formation of supernumerary units very often lead to dystopia. This is due to the fact that the space on the dental arch line is limited, and the roots of normal teeth simply push the “intruder” towards the cheek or palate.
  • Impacted teeth – impaction occurs when a tooth loses its growth impulse and remains embedded in the jawbone. Impacted teeth can cause the adjacent normal teeth to become loose and cause them to shift and change the bite. Often cause pain.

On a note! According to statistics, hyperdontia accounts for up to 2% of cases of dental problems. Of these, 70% are associated with the appearance of single supernumerary teeth, 25% with a couple of such formations, and only in 5% complex multiple complexes of 3–4 or more teeth can be found.

What is polyodontia?

Supernumerary teeth are extra units in the dentition. There can be 33,34,36 or even more than 100. Normally, the number of teeth in a child’s primary occlusion should not exceed 20, and in an adult – 32 units.

Most often, additional incisors or canines appear. It is much less common for a person to grow extra wisdom teeth, and they usually remain impacted.

Supernumerary teeth in an adult differ from the rest in size, development of the crown and root. They can also have a different shape, resembling, for example, a thorn or a drop.

If the extra units have not erupted, then polyodontia usually does not cause any unpleasant sensations. Therefore, we often learn about the existence of supernumerary teeth only during radiography.

Diagnostic methods


The main indication for diagnosis is the presence of an “extra” tooth in the patient’s oral cavity or obvious signs of its eruption (painful tubercle or swelling in the gum or palate). All of these signs can be identified through a routine visual examination, during which the dentist evaluates the condition of the oral cavity. To confirm the diagnosis and carry out differential diagnosis, an X-ray examination is performed - an orthopantomogram. If it is necessary to examine the dentition in several planes, computed tomography (CT) is additionally prescribed.

On a note! The patient may not be aware of the presence of impacted supernumerary teeth. In this case, hyperdontia is detected only by the results of an X-ray or CT scan of the maxillofacial region.

Removal of impacted teeth

In order for the operation to be successful and polyodontia to be cured without any complications, the doctor must fully examine the patient and plan his further actions.

  • To begin with, X-rays and/or computed tomography are performed to determine the exact topography of the anomaly.
  • Removal is performed under local anesthesia, but there are cases when general anesthesia can be used on the patient.
  • First, the mucous membrane is peeled off, then the bone tissue is opened and the root and crown parts of the tooth are removed.
  • If necessary, bone defects are covered with osteoplastic material, and the mucous membrane is sutured.

After tooth extraction, the patient continues treatment at home: takes antibiotics (if prescribed by the attending physician), rinses the oral cavity with antiseptic solutions.

Until the wound heals after surgery, it is not recommended to eat too hot, hard or spicy food. You should also brush your teeth carefully, especially on the operated side.

Do supernumerary teeth need to be removed?

There is no clear answer to the question of what to do with “extra” teeth. Much depends on the shape, position and quantity. The following are subject to mandatory removal:

  • baby teeth that interfere with normal growth and formation of permanent teeth;
  • strongly dystopic structures - located on the palate or at a large angle to the lateral side of the gums;
  • impacted formations that put pressure on the adjacent roots of normal teeth and provoke periodic inflammatory processes of soft tissues.

If the additional tooth does not cause discomfort and does not disrupt the development of the dentition, removal may not be necessary. Moreover, such a superset can play the role of a strategic reserve in case of damage to nearby normal teeth.

How does the removal work?

The removal procedure can be simple or complex. In the first case, the doctor is dealing with a fully erupted tooth, the root of which is not intertwined with the surrounding structures. Simple removal steps:

  • The oral cavity is prepared - treated with antiseptics, anesthesia is given.
  • Grasp the crown with forceps (in some cases, an additional incision of the mucous tissue is required to facilitate access).
  • The elevator destroys the retaining ligaments.
  • The root is freed and the tooth is removed.
  • Clean the hole from tooth fragments and bones.
  • Apply stitches (if necessary) and a healing bandage.

Complex extraction is prescribed in the case of impacted, semi-impacted or dystopic teeth with complex root shapes. The procedure is carried out with a deep incision of soft tissues, in especially severe cases - with opening of the jaw bone to gain access to the roots. The latter option is relevant for deep bone occurrence, position in close proximity to the cranial sinuses and orbits, as well as for irregularly shaped roots and a high risk of damage to complete teeth. A complex removal operation is performed by a dental surgeon.

ICD-10 (Dentistry)

​The article presents the international classification of diseases, tenth revision, relating to the dental profile.

K00—K14 Diseases of the oral cavity, salivary glands and jaws (click on the appropriate block to expand subcategories)
K00 Disorders of development and eruption of teeth
Excluding: impacted and impacted teeth (K01)

K00.0Edentia
  • hypodontia
  • oligodontia
K00.1Supernumerary teeth
  • distomolar teeth
  • fourth molar
  • mesiodentia (middle tooth)
  • paramolar teeth
  • extra teeth
K00.2Anomalies in the size and shape of teeth
  • fusion of teeth
  • dental fusion
  • germination of teeth
  • protrusion of teeth, “tooth in tooth”, invagination of teeth
  • enamel pearls
  • macrodentia
  • microdentia
  • spear-shaped (conical) teeth
  • "bull's tooth"
  • paramolar accessory cusps

Excluding: Carabelli tubercular anomaly, considered as a normal variant and subject to coding

K00.3Mottled teeth
  • dental fluorosis
  • enamel mottling
  • non-fluorotic darkening of enamel

Excluding: deposits (growths) on teeth (K03.6)

K00.4Tooth formation disorders
  • aplasia and hypoplasia of cement
  • enamel cracks
  • enamel hypoplasia (neonatal, postnatal, prenatal)
  • regional odontodysplasia
  • Turner teeth

Excludes: Hutchinson's incisors and mulberry-shaped molars in congenital syphilis (A50.5), mottled teeth (K00.3)

K00.5Hereditary disorders of dental structure, not classified elsewhere
  • underdevelopment of enamel
  • underdevelopment of dentin
  • underdevelopment of the tooth
  • dentin dysplasia
  • conch teeth
K00.6Teething disorders
  • early teething
  • Natal teeth (erupted at birth)
  • neonatal teeth (in a newborn, erupted prematurely)
  • premature eruption, loss of primary (temporary) teeth
  • delayed change of primary teeth
K00.7Teething syndrome
K00.8Other dental development disorders
  • change in tooth color during formation
  • pronounced staining of teeth NOS (no further specification)
K00.9Dental development disorder, unspecified
  • odontogenesis disorder NOS (not otherwise specified)

K01 Impacted and impacted teeth
Excluding: impacted and impacted teeth with malposition of them or adjacent teeth (K07.3)

K01.0Impacted teeth
An impacted tooth is a tooth that has changed its position during eruption without obstruction from an adjacent tooth.
K01.1Impact teeth
An impact tooth is a tooth that has changed its position during eruption due to an obstacle from an adjacent tooth.

K02 Dental caries

K02.0Enamel caries
  • “chalk spot” stage (initial caries)
K02.1Dentin caries
K02.2Cement caries
K02.3Suspended dental caries
K02.4Odontoclasia
  • childhood melanodentia
  • melanodontoclasia
K02.8Other dental caries
K02.9Dental caries, unspecified

K03 Other diseases of hard dental tissues
Excluding: bruxism, teeth grinding NOS (not otherwise specified) (F45.8), dental caries (K02)

K03.0Increased tooth wear
  • tooth abrasion: aproximal, occlusal
K03.1Grinding of teeth
  • grinding of teeth: caused by tooth powder
  • habitual
  • professional
  • ritual
  • traditional
  • wedge-shaped defect NOS (no further specification)
K03.2Tooth erosion
  • due to: diet
  • medicines and medicines
  • permanent job
  • idiopathic
  • professional
  • NOS (no further details)
K03.3Pathological tooth resorption
  • internal pulp granuloma
  • resorption of hard dental tissues (external)
K03.4Hypercementosis
  • cementum hyperplasia
K03.5Ankylosis of teeth
K03.6Deposits (growths) on teeth
  • subgingival and supragingival calculus
  • deposits (growths) on teeth: betel nuts
  • black
  • green
  • white
  • orange
  • tobacco
  • teeth staining
K03.7Change in color of hard tissues of teeth after eruption
Excluding: deposits (growths) on teeth (K03.6)
K03.8Other specified diseases of dental hard tissues
  • irradiated enamel
  • sensitive dentin

If it is necessary to identify the radiation that caused the injury, use an additional code of external causes (class XX).

K03.9Disease of hard dental tissues, unspecified

K04 Diseases of the pulp and periapical tissues

K04.0Pulpitis
  • pulp abscess and polyp
  • pulpitis: acute
  • chronic (hyperplastic, ulcerative)
  • purulent
K04.1Pulp necrosis
  • pulp gangrene
K04.2Pulp degeneration
  • denticles
  • pulp calcifications and stones
K04.3Improper formation of hard tissue in the pulp
  • formation of secondary, or irregular, dentin
K04.4Acute apical periodontitis of pulpal origin
  • acute apical periodontitis NOS (not otherwise specified)
K04.5Chronic apical periodontitis
  • apical or periapical granuloma
  • apical periodontitis NOS (not otherwise specified)
K04.6Periapical abscess with cavity
  • dental abscess with cavity
  • dentoalveolar abscess with cavity
K04.7Periapical abscess without cavity
  • dental abscess NOS (not otherwise specified)
  • dentoalveolar abscess NOS (not otherwise specified)
  • periapical abscess NOS (not otherwise specified)
K04.8Root cyst
  • apical (periodontal) cyst
  • periapical cyst
  • residual root cyst

Excludes: periodontal lateral cyst (K09.0)

K04.9Other and unspecified diseases of the pulp and periapical tissues

K05 Gingivitis and periodontal diseases

K05.0Acute gingivitis
Excludes: acute necrotizing ulcerative gingivitis (A69.1), gingivostomatitis caused by herpes simplex virus (B00.2)
K05.1Chronic gingivitis
  • desquamative
  • hyperplastic
  • simple marginal
  • ulcerative
  • NOS (no further details)
K05.2Acute periodontitis
  • acute pericoronitis
  • periodontal abscess
  • periodontal abscess

Excluding:

  • acute apical periodontitis of pulpal origin (K04.4)
  • periapical abscess (K04.7)
  • periapical abscess with cavity (K04.6)
K05.3Chronic periodontitis
  • chronic pericoronitis
  • periodontitis: simple
  • difficult
  • NOS (no further details)
K05.4Periodontal disease
  • juvenile periodontal disease
K05.5Other periodontal diseases
K05.6Periodontal disease, unspecified

K06 Other changes in the gingiva and edentulous alveolar margin
Excluding: atrophy of the edentulous alveolar margin (K08.2), gingivitis: acute (K05.0), chronic, NOS (not otherwise specified) (K05.1)

K06.0Gum recession
  • gum recession (generalized, local, post-infectious, post-operative)
K06.1Gingival hypertrophy
  • gingival fibromatosis
K06.2Lesions of the gums and edentulous alveolar margin caused by trauma.
If necessary, identify the cause, use an additional code for external causes (class XX)
K06.8Other specified changes in the gingiva and edentulous alveolar margin
  • fibrous epulis
  • atrophic ridge
  • giant cell epulis
  • giant cell peripheral granuloma
  • pyogenic granuloma of the gums
K06.9Changes in the gingiva and edentulous alveolar margin, unspecified

K07 Maxillofacial anomalies (including malocclusions)
Excludes: atrophy and hypertrophy of the half of the face (Q67.4), unilateral condylar hyperplasia or hypoplasia (K10.8)

K07.0Main anomalies in jaw size
  • hyperplasia, hypoplasia (lower and upper jaw)
  • macrognathia (lower and upper jaw)
  • micrognathia (lower and upper jaw)

Excludes: acromegaly (E22.0), Robin's syndrome (Q87.0)

K07.1Anomalies of maxillo-cranial relationships
  • jaw asymmetry
  • prognathia (lower and upper jaw)
  • retrognathia (lower and upper jaw)
K07.2Anomalies of dental arch relationships
  • displaced bite (anterior, posterior)
  • distal bite
  • mesial bite
  • displacement of dental arches from the midline
  • open bite (anterior, posterior)
  • overbite: deep
  • horizontal
  • vertical
  • fan-shaped bite
  • posterior lingual bite of the lower teeth
K07.3Anomalies of teeth position
  • crowding of the tooth(s)
  • diastema of the tooth (teeth)
  • displacement of tooth(s)
  • rotation of tooth(s)
  • violation of interdental spaces
  • transposition of tooth(s)
  • Impacted or impacted teeth with improper positioning of them or adjacent teeth

Excludes: impacted and impacted teeth with normal position (K01)

K07.4Malocclusion, unspecified
K07.5Maxillofacial anomalies of functional origin
  • improper jaw closure
  • malocclusion: due to disturbances in swallowing, mouth breathing, tongue, lip or finger sucking

Excludes: bruxism, teeth grinding NOS (not otherwise specified) (F45.8)

K07.6Temporomandibular joint diseases
  • syndrome, or complex, Kosten
  • Looseness of the temporomandibular joint
  • "clicking" jaw
  • temporomandibular joint pain dysfunction syndrome

Excludes: current case of jaw dislocation (S03.0), sprain and strain of jaw joint(s) (S03.4)

K07.8Other maxillofacial anomalies
K07.9Maxillofacial anomaly, unspecified

K08 Other changes in teeth and their supporting apparatus

K08.0Exfoliation of teeth due to systemic disorders
K08.1Loss of teeth due to accident, extraction or localized periodontal disease
K08.2Atrophy of the edentulous alveolar margin
K08.3Delayed tooth root (retentive root)
K08.8Other specified changes in teeth and their supporting apparatus
  • hypertrophy of the alveolar margin NOS (not otherwise specified)
  • irregular shape of the alveolar process
  • toothache NOS (not otherwise specified)
K08.9Changes in teeth and their supporting apparatus, unspecified

K09 Cysts of the oral region, not elsewhere classified
Including: lesions with histological features of an aneurysmal cyst and other fibro-osseous lesion Excluding: radicular cyst (K04.8)

K09.0Cysts formed during the formation of teeth
  • cyst: containing teeth
  • formed during teething
  • follicular
  • gums
  • lateral periodontal
  • rudimentary
  • horny cyst
K09.1Growth (non-odontogenic) cysts of the mouth area
  • globulomaxillary cyst (maxillary sinus cyst)
  • incisor canal cyst
  • midpalatal cyst
  • nasopalatine cyst
  • palatine papillary cyst
K09.2Other jaw cysts
  • jaw cyst: aneurysmal, hemorrhagic, traumatic, NOS (not otherwise specified)

Excludes: occult bone cyst of the jaw, Stafne cyst (K10.0)

K09.8Other specified cysts of the oral area, not classified elsewhere
  • oral dermoid cyst
  • epidermoid cyst of the oral cavity
  • lymphoepithelial cyst of the oral cavity
  • Epstein's pearl
  • nasoalveolar cyst
  • nasolabial cyst
K09.9Oral cyst, unspecified

K10 Other jaw diseases

K10.0Jaw development disorders
  • hidden bone cyst of the jaw
  • Stafne cyst
  • torus of the mandible and hard palate
K10.1Giant cell granuloma, central
  • giant cell granuloma NOS (not otherwise specified)

Excludes: peripheral giant cell granuloma (K06.8)

K10.2Inflammatory diseases of the jaws
  • Osteitis of the jaw (acute, chronic, purulent)
  • osteomyelitis (neonatal) of the jaw (acute, chronic, purulent)
  • radiation osteonecrosis of the jaw (acute, chronic, purulent)
  • periostitis of the jaw (acute, chronic, purulent)
  • jawbone sequestration

If necessary, identify the radiation that caused the injury, use an additional code of external causes (class XX)

K10.3Alveolitis of the jaws
  • alveolar osteitis
  • dry socket
K10.8Other specified diseases of the jaws
  • Cherubism
  • exostosis of the jaw
  • fibrous dysplasia of the jaw
  • unilateral condylar hyperplasia, unilateral condylar hypoplasia
K10.9Disease of the jaw, unspecified

K11 Diseases of the salivary glands

K11.0Salivary gland atrophy
K11.1Salivary gland hypertrophy
K11.2Sialadenitis
Excludes: mumps (B26), Hereford uveoparotid fever (D86.8)
K11.3Salivary gland abscess
K11.4Salivary gland fistula
Ex: congenital salivary gland fistula (Q38.4)
K11.5Sialolithiasis
  • salivary gland or duct stones
K11.6Salivary gland mucocele
  • mucous cyst of the salivary gland with exudate, mucous retention cyst of the salivary gland
  • ranula
K11.7Disorders of the secretion of the salivary glands
  • hypoptialism
  • ptyalism
  • xerostomia

Excludes: dry mouth NOS (R68.2)

K11.8Other diseases of the salivary glands
  • benign lymphoepithelial lesion of the salivary gland
  • Mikulicz's disease
  • necrotizing sialometaplasia
  • sialectasia
  • salivary duct stenosis
  • narrowing of the salivary duct

Excludes: sicca syndrome (Sjögren's disease) (M35.0)

K11.9Salivary gland disease, unspecified
  • sialoadenopathy NOS (not otherwise specified)

K12 Stomatitis and related lesions
Excluding:

  • decaying mouth ulcer, gangrenous stomatitis, noma (A69.0)
  • cheilitis (K13.0)
  • gingivostomatitis caused by herpes simplex virus (B00.2)
K12.0Recurrent oral aphthae
  • aphthous stomatitis (large, small)
  • Bednar's aphthae
  • recurrent muconecrotizing periadenitis
  • recurrent aphthous ulcer
  • stomatitis herpetiformis
K12.1Other forms of stomatitis
  • dental stomatitis
  • ulcerative stomatitis
  • vesicular stomatitis
  • stomatitis NOS (not otherwise specified)
K12.2Cellulitis and oral abscess
  • inflammation of the tissue of the oral cavity (bottom)
  • abscess of the submandibular region

Excluding:

  • periapical abscess (K04.6—K04.7)
  • periodontal abscess (K05.2)
  • peritonsillar abscess (J36)
  • salivary gland abscess (K11.3)
  • tongue abscess (K14.0)

K13 Other diseases of the lips and oral mucosa
Including: changes in the epithelium of the tongue Excluding:

  • some changes in the gingiva and edentulous alveolar margin (K05-K06)
  • cysts of the mouth area (K09)
  • tongue diseases (K14)
  • stomatitis and related lesions (K12)
K13.0Lip diseases
  • angular cheilitis
  • exfoliative
  • glandular
  • NOS (no further details)
  • cheilodynia
  • cheilosis
  • lip commissure fissure (jam) NOS (not otherwise specified)
  • Excluding:

    • ariboflavinosis (E53.0)
    • Radiation-associated cheilitis (L55–L59)
    • fissure of the commissure of the lips (jam): due to candidiasis (B37.8), due to riboflavin deficiency (E53.0)
K13.1Biting cheeks and lips
K13.2Leukoplakia and other changes in the oral epithelium, including the tongue
  • erythroplakia of the oral epithelium, including the tongue
  • leukedema of the oral epithelium, including the tongue
  • nicotinic leukokeratosis of the palate
  • smoker's sky

Excludes: hairy leukoplakia (K13.3)

K13.3Hairy leukoplakia
K13.4Granuloma and granuloma-like lesions of the oral mucosa
  • eosinophilic granuloma of the oral mucosa
  • pyogenic granuloma of the oral mucosa
  • verrucous xanthoma of the oral mucosa
K13.5Submucosal fibrosis of the oral cavity
  • submucosal fibrosis of the tongue
K13.6Hyperplasia of the oral mucosa due to irritation
Excludes: hyperplasia of the edentulous alveolar margin due to irritation (denture hyperplasia) (K06.2)
K13.7Other and unspecified lesions of the oral mucosa
  • focal oral mucinosis

K14 Diseases of the tongue
Excluding:

  • erythroplakia, focal epithelial hyperplasia, leukedema, leukoplakia of the tongue (K13.2)
  • hairy leukoplakia (K13.3)
  • congenital macroglossia (Q38.2)
  • submucosal fibrosis of the tongue (K13.5)
K14.0Glossitis
  • tongue abscess
  • ulceration of the tongue (traumatic)

Excludes: atrophic glossitis (K14.4)

K14.1"Geographical" language
  • benign migratory glossitis
  • exfoliative glossitis
K14.2Median rhomboid glossitis
K14.3Hypertrophy of the tongue papillae
  • glossophytia ("black hairy tongue")
  • coated tongue
  • hypertrophy of foliate papillae
  • lingua villosa nigra
K14.4Atrophy of the tongue papillae
  • atrophic glossitis
K14.5Folded tongue
  • cleft tongue
  • grooved tongue
  • wrinkled tongue

Excludes: congenital cleft tongue (Q38.3)

K14.6Glossodynia
  • burning sensation in tongue
  • glossalgia
K14.8Other tongue diseases
  • tongue atrophy
  • serrated tongue
  • enlarged tongue
  • hypertrophied tongue
K14.9Tongue disease, unspecified
  • glossopathy NOS (not otherwise specified)

Dental diseases from other sections.

A69.0Necrotizing ulcerative stomatitis
  • Gangrenous stomatitis
  • Fusospirochetous gangrene
  • Noma
  • Fast-breaking oral ulcers
A69.1Other Vincent infections - Fusospirochetous pharyngitis - Necrotizing ulcerative (acute): • gingivitis • gingivostomatitis - Spirochetal stomatitis - Vincent's ulcerative film sore throat: • tonsillitis • gingivitis
B00.2Herpetic gingivostomatitis and pharyngotonsillitis
B26Parotitis:
  • Mumps orchitis
  • Mumps meningitis
  • Mumps encephalitis
  • Mumps pancreatitis
  • Mumps with other complications
  • Mumps, uncomplicated
B37.8Candidiasis of other localizations
Candidiasis: • cheilitis • enteritis
D86.8Sarcoidosis of other specified and combined localizations:
  • Iridocyclitis in sarcoidosis
  • Multiple cranial nerve palsies in sarcoidosis
  • Sarcoid: arthropathy, myocarditis, myositis
  • Uveoparotitic fever, Herfordt's disease
E22.0Acromegaly and pituitary gigantism
Excluded:
  1. hypersecretion of growth hormone releasing hormone
  2. constitutional:
  • high growth
  • gigantism
E53.0Riboflavin deficiency
  • Ariboflavinosis
F45.8Any other disturbances in sensation, function, and behavior that are not associated with physical disorders and that are not mediated through the autonomic nervous system are limited to specific systems or parts of the body and are closely related in time to stressful events or problems. Psychogenic:
  • dysmenorrhea
  • dysphagia, including "globus hystericus"
  • itching
  • torticollis
  • teeth grinding
J36Peritonsillar abscess
  • Tonsil abscess
  • Peritonsillar cellulitis
  • Quincy

Excluded:

  • retropharyngeal abscess
  • tonsillitis: • NOS (not otherwise specified) • acute [tonsillitis] • chronic
L55—L59Diseases of the skin and subcutaneous tissue associated with exposure to radiation
  • L55 Sunburn
  • L56 Other acute skin changes caused by ultraviolet radiation
  • L57 Skin changes caused by chronic exposure to non-ionizing radiation
  • L58 Radiation dermatitis
  • L59 Other diseases of the skin and subcutaneous tissue associated with radiation
M35.0Sicca Sjögren's syndrome
Sjögren's syndrome with:
  • keratoconjunctivitis
  • lung damage
  • myopathy
  • tubulointerstitial kidney damage
Q38.2Macroglossia
Q38.3Other congenital abnormalities of the tongue
  • Aglossia
  • Forked tongue
  • Congenital: • tongue commissure • tongue fissure • tongue anomaly NOS
  • Hypoglossia
  • Tongue hypoplasia
  • Microglossia
Q38.4Congenital anomalies of the salivary glands and ducts:
  • Absence of salivary gland or duct
  • Accessory salivary gland
  • Atresia of the salivary gland or duct
  • Congenital fistula of the salivary gland
Q67.4Other congenital deformities of the skull, face and jaw
Q87.0Syndromes of congenital anomalies affecting primarily the appearance of the face
  • Acrocephalopolysyndactyly
  • Acrocephalosyndactyly [Aperta]
  • Cryptophthalmos syndrome
  • Cyclopia
  • Goldenhar syndrome
  • Mobius syndrome
  • oro-facial-digital syndrome
  • Robin syndrome
  • Treacher Collins
  • The face of a whistling man
R68.2Dry mouth, unspecified
Excluded:

decreased secretion of salivary glands (K11.7)

dry mouth caused by:

  • dehydration
  • sicca [Sjögren's] syndrome
S03.0Jaw dislocation:
  • Jaw (cartilage) (meniscus)
  • Lower jaw
  • Temporomandibular joint
S03.4Sprain and strain of the joint (ligaments) of the jaw
Temporomandibular joint (ligament)
class XXExternal causes of morbidity and mortality:
  • Pedestrian injured in a traffic accident
  • Cyclist injured in a traffic accident
  • Motorcyclist injured in a traffic accident
  • Occupant of a three-wheeled motor vehicle injured in a traffic accident
  • A person who was in a car and was injured as a result of a transport accident
  • An occupant of a pickup truck or van who is injured in a transportation accident
  • Person who was in a heavy truck and was injured as a result of a transport accident
  • A person on a bus who was injured in a traffic accident
  • Accidents involving other land vehicles
  • Water transport accidents
  • Accidents in air transport and space flights
  • Other and unspecified transport accidents
  • Falls
  • Impact of non-living mechanical forces
  • Impact of living mechanical forces
  • Accidental drowning and submersion
  • Other respiratory hazards
  • Accidents caused by electrical current, radiation and extreme levels of ambient temperature or atmospheric pressure
  • Exposure to smoke, fire and flames
  • Contact with hot and incandescent substances (objects)
  • Contact with poisonous animals and plants
  • Impact of the forces of nature
  • Accidental poisoning and exposure to toxic substances
  • Overexertion, travel and hardship
  • Accidental exposure to other and unspecified factors
  • Deliberate self-harm
  • Attack
  • Damage with uncertain intent
  • Legal actions and military operations
  • Drugs, medications and biological substances that cause adverse reactions during therapeutic use
  • Accidental harm to a patient during therapeutic and surgical interventions
  • Medical devices and devices associated with accidents arising from their use for diagnostic and therapeutic purposes
  • Surgical and other medical procedures as the cause of an abnormal response or late complication in a patient without mention of accidental harm during their performance
  • Consequences of external causes of morbidity and mortality

Orthodontic treatment

Depending on the situation, it is used after tooth extraction or instead of it. In children, it is used to stimulate the loss of baby teeth and eliminate false hyperdontia, as well as to facilitate and accelerate the eruption of a normal set. In addition, orthodontic treatment with the installation of braces, mouth guards, dental plates or other auxiliary structures can straighten crooked teeth and return them to their proper place.

Before prescribing treatment, the dentist carefully evaluates the condition of the complete and supernumerary structures and, depending on the overall picture and prognosis for the future, can use both formations or leave the strongest and most durable tooth (not necessarily complete).

Normal deletion

If the dentist decides that in a particular case, polyodontia can only be treated by removing an extra tooth, the patient should count on the following procedures:

  • First of all, the patient should be sent for radiography. This is necessary in order to determine the size and number of roots, as well as the ratio of supernumerary and normal teeth.
  • After collecting research, the doctor gives the patient anesthesia and removes excess teeth.
  • In some cases, soft tissue sutures may be necessary after surgery.

Classification of injuries to children's teeth

The nature and severity of the damage is determined based on a comprehensive examination of the patient using radiological, clinical and other methods. To make a diagnosis, dentists today use different systems for diagnosing trauma to children’s teeth.

Standard ELLIS classification

This system of dividing acute injuries into separate types, developed by Ellis, is the very first and most used in practice. It includes 9 categories of dental damage:

  • I – slight fracture of the crown within the tooth enamel;
  • II – moderate crown fracture extending into enamel and dentin;
  • III – severe fracture of the crown with exposure of the pulp;
  • IV – loss of pulp viability in the presence/absence of a crown fracture;
  • V – complete dislocation of the injured tooth;
  • VI – damage with root fracture;
  • VII – slight subluxation of an injured tooth;
  • VIII – fracture of the crown in the cervical area;
  • IX – trauma to baby teeth.

Types of Traumatic Injuries WHO

Actively used in modern dentistry along with ELLIS. This classification was developed based on the Andresen system and includes, in addition to dental injuries, damage to other tissues (oral mucosa, gums, periodontal tissue, bones).

I. Damage to pulp and hard tissue:

  • cracks in tooth enamel (A);
  • fracture of the crown without opening the pulp (enamel affected - B, enamel and dentin affected - C);
  • fracture of the dental crown exposing the pulp (D);
  • combined root and crown fracture with pulp exposure (E);
  • trauma with root fracture (F).

II. Periodontal damage:

  • shock of the tooth with a weak response of periodontal tissue to percussion without increasing its mobility and displacement (A);
  • subluxation with change in mobility without displacement (B);
  • penetration of the injured tooth deep into the tissues (C);
  • exit of the tooth from the anatomical socket (D);
  • displacement in a direction other than axial (E);
  • complete dislocation of the injured tooth (F).

III. Bone injury:

  • tight connection of the tooth with the socket with penetration deep into the tissue (A);
  • fracture of the wall of the anatomical recess (B);
  • fracture of the alveolar bone tissue (C, D);
  • fracture of the jaw bone (upper – E, lower – F).

IV. Injury to the gums and oral mucosa:

  • acute soft tissue injury (A);
  • bruise of the mucous membrane or gums (B);
  • soft tissue detachment (C).

Classification of injuries to children's teeth according to ICD-C

This is an international system for defining acute injuries, based on ICD-10 (1997). Used in pediatric dentistry to classify various injuries, including fractures of primary and permanent teeth (S02.5):

  • fracture (chip) within the thickness of the enamel (S02.50);
  • fracture of the dental crown without injury to the pulp (S02.51);
  • crown fracture with injury extending to the pulp (S02.52);
  • fracture of the root part of a permanent tooth (S02.53);
  • double fracture in the area of ​​the dental crown and root (S02.54);
  • multiple fractures of different locations (S02.57);
  • unspecified fracture of primary or permanent tooth (S02.59).

Procedure for providing assistance to children with dental damage


Immediately after receiving an injury, the patient is provided with primary medical care, which includes a general assessment of the child’s condition, pain relief, prescription of antibiotics, anesthetics and other medications, as well as a preliminary diagnosis. After this, the patient’s parents are given a recommendation to make an appointment with a pediatric dentist (therapist), who will carry out full dental treatment. The doctor providing specialized care carries out:

  • registration of a medical history (including legal and social aspects of the case);
  • collecting anamnesis (origin of injury, presence of concussion, local symptoms);
  • clinical examination of the injury (visual examination, percussion, palpation, temperature tests of the pulp, instrumental methods);
  • additional diagnostics (electroodontodiagnostics (EDD), transillumination, targeted radiography, occlusal radiography, computed tomography);
  • making a diagnosis based on examination, clinical and other studies;
  • choice of treatment tactics taking into account the nature of the damage, possible risks, benefits and costs.

The period of rehabilitation of a child after an injury, starting from the moment of emergency assistance, can take 1-3 or more days. Given the complexity of treatment, the period of complete restoration of the integrity and functions of damaged teeth often takes a longer period - several months or even years.

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